How bone responds to load
Bone is not scaffolding. It is living tissue that gets taken apart and rebuilt continuously, and on average your whole skeleton is replaced every eight to ten years [Osteoporosis NZ, 2024]. What decides whether it comes back thicker or thinner is largely what you asked it to carry.
The Royal Osteoporosis Society puts it in two sentences: “If you are not active, your bones lose strength. Bones get stronger when you use them.” [Royal Osteoporosis Society, 2026]
Two forces do that work. One is muscle: as a muscle contracts it pulls on the bone it is attached to, and that pull is what maintains or improves the bone's strength. The other is impact, the force that travels up through the skeleton each time a foot hits the ground [Royal Osteoporosis Society, 2026]. Lifting supplies the first. Walking, stamping and stair climbing supply the second.
The response is local. Bone that gets loaded responds; bone that does not, does not. The 2022 UK consensus statement on exercise for osteoporosis describes the effects of exercise on bone as “highly localised,” and says resistance exercises should be chosen to load “skeletal sites at risk of osteoporotic fracture, such as the spine, proximal femur and forearm” [BJSM, 2022]. Spine, hip, wrist. That is also why a daily walk, good as it is for the hips, does almost nothing for a wrist.
Until your mid-twenties, bone formation outruns breakdown. From then to about 50 the two roughly match [Osteoporosis NZ, 2024]. From 50 onward breakdown wins, and in women it speeds up through menopause as oestrogen falls [Osteoporosis NZ, 2024].
None of that closes the door. Loading still changes bone at 68. It just has to be put there on purpose, because ordinary daily life stopped supplying enough of it years ago.